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[We now show that p57 is expressed predominantly in the subcommissural organ and cerebellar interneurons in the mouse brain and that mice with brain-specific deletion of the p57 gene (Kip2) manifest prominent nonobstructive hydrocephalus as well as cerebellar malformation associated with the loss of Pax2-positive interneuron precursors and their descendants, including Golgi cells and ?-aminobutyric acid-containing neurons of the deep cerebellar nuclei.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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